Wernicke Encephalopathy After Bariatric Surgery Objective: To dental review the clinical foundations of Wernicke encephalopathy (WE), after bariatric surgery. Summary center Background Data: An estimated 205,000 bariatric department surgical insurance procedures were performed in the United States in 2007. Such procedures can lead to serious complications of nutrition. Methods: We searched community bibliographies of Medline, EMBASE, and abstract collections. Inclusion criteria were WE after bariatric surgery, diagnosed by the presence of two or more of the following symptoms: mental status changes, ocular movement abnormalities, cerebellar dysfunction, and nutrition poor diet. Results: Of 104 cases reported of WE after bariatric surgery, 84 cases were included. Gastric bypass or restrictive procedure has been performed in 80 cases (95 ). the best insurance plan can be found with has cost-effective health plan solutions Admission to hospital for U.S. produced within 6 months of surgery in 79 cases (94 ). Vomiting was often a risk factor in 76 cases (90 ) and lasted an average of 21 days at admission. Intravenous glucose without thiamine was a risk factor in 15 clinic cases (18 ). Magnetic resonance imaging of the brain identified characteristic lesions of the week in 14 of 30 cases (47 ). Incomplete recovery was observed in 41 cases (49 ), medical memory deficits and difficulties of travel were common hospital sequelae. The recent weight loss increased use of bariatric surgery in the United States was associated with an increase in cases of WE. benefits Conclusions: The natural number of cases of WE after bariatric surgery is substantially higher than previously reported. Surgeons, associated health providers and patients should be aware of the predisposing factors and symptoms to prevent and optimize the management of this condition Source: Medscape Surgery. December 2008 Version extensively with this article